Treating seizures in renal and hepatic failure
نویسندگان
چکیده
منابع مشابه
Congenital Hepatic Fibrosis: An Uncommon Cause of Chronic Renal Failure
Congenital Hepatic Fibrosis (CHF) is a rare disease that affects both the liver and kidneys. Congenital hepatic fibrosis (CHF) is an autosomal recessive inherited malformation defined pathologically by a variable degree of periportal fibrosis and irregularly shaped proliferating bile ducts. Affected individuals also have impaired renal function, usually caused, in children and teenagers, by an...
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Eighteen patients with fulminant liver failure were studied, 10 with normal renal function (group A) and eight with renal failure (group B, plasma creatinine greater than 200 mumol/l). Renal function was assessed by standard clearance techniques and patients in group B had a marked reduction compared with group A in both renal plasma flow and glomerular filtration rate. Raised plasma renin acti...
متن کاملRenal failure in children with hepatic failure undergoing liver transplantation.
Over a 3 1/2 year period, 133 children with hepatic failure underwent orthotopic liver transplantation (OLT) at our center. Renal failure (creatinine clearance less than 20 ml/min/1.73 m2) was present in 19 (14.3%) of these children. In seven of the 19 children, renal failure was present before OLT, and in the other 12 after OLT. The causes of renal failure included hepatorenal syndrome in seve...
متن کاملSpectrum of renal tubular damage in renal failure secondary to cirrhosis and fulminant hepatic failure.
Measurements of urinary lysozyme were used to evaluate renal tubular integrity in 34 patients with cirrhosis or fulminant hepatic failure who had developed renal impairment. In 18 of the patients the lysozyme values were normal but in the remaining 16 were increased, supporting previous concepts that renal failure complicating hepatocellular disease may occur both without and with tubular necro...
متن کاملSeizures and renal failure: is there a link?
A 70-year-old woman was referred to the emergency room of our hospital because of asthenia and polypnea. On physical examination, the patient was a slender woman who appeared to be afebrile and clinically hypovolaemic. Her vital signs were normal; there was no pedal oedema, blood pressure was 130/90mmHg, and body weight 56 kg. The levels of serum creatinine and urea nitrogen were 6 and 180mg/dl...
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ژورنال
عنوان ژورنال: Journal of Epilepsy and Clinical Neurophysiology
سال: 2008
ISSN: 1676-2649
DOI: 10.1590/s1676-26492008000600008